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Recent in Skin Science (page 12 of 29)
Daniel Popkin, MD, and colleagues were awarded $25,000 by the NRS to study the facial microbiomes of identical twins in whom only one has rosacea.
July 2015 conference aims at increasing understanding of psoriasis and psoriatic arthritis in order to improve patient outcomes.
A newly discovered mechanism—EphA2 protein activity—may lead to drug resistance and metastasis in melanoma patients. Researchers are investigating strategies to counteract this novel mechanism.
Until now, a melanoma's rampant growth was mainly attributed to genetic causes, however, researchers from the University of Zurich now reveal that so-called epigenetic factors play a role.
1/8/2015 | Erin Madigan-Fleck
Research and statistics have demonstrated a commonality in the link between autoimmune disease and the correlation of skin symptoms, which may greatly impact esthetic care.
1/8/2015 | Robert P. Manzo
Why is it that a cosmeceutical works so well on one client for reducing wrinkles, redness and pore size, but does not work at all for another? The answer is bioavailability.
Free radicals might not be all bad, according to researchers from the University of California, San Diego. Andrew Chishold, PhD, and his fellow researchers found reactive oxygen species (ROS) to be beneficial to wound healing.
A scientific team at UBC and Providence Health Care have genetically engineered mice with less wrinkled skin, despite repeated exposure to wrinkle-inducing ultraviolet (UV) light.
Eczema was not associated overall with short stature in an analysis of data from several studies, according to a study published online by JAMA Dermatology.
A piece of research has concluded that skin depigmentation in Europeans has taken place through an adaptive process furthered by natural selection.
12/8/2014 | Annette Tobia, PhD
To be direct: Sugar kills skin. It can turn an ivory-smooth complexion into a lunar surface of wrinkled, crépey, irritated skin. And this can happen quickly.
Inflammatory skin diseases, such as psoriasis, may result from abnormal activation of cell death pathways previously believed to suppress inflammation, a surprise finding that could help to develop new ways of treating these diseases.